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To summarize these data, our model of acute BEC death displays the classical histopathology observed in patients with cholangiopathies, including portal inflammation, fibrosis, and BEC proliferation, as well as cholestasis and portal hypertension, and represents what we believe to be a good model to study the early signals that drive bile duct repair
Kollagen
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When • X was generated at C-3 and C-4′ OH positions of quercetin, there was a greater delocalization of the unpaired electron, yielding a greater number of resonant forms (in blue and green)
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