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Patient care services are rendered and documented in the patient’s medical record
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Not only β-catenin can affect the levels of p53, but p53 can in turn modulate the levels of β-catenin, enhancing its proteasomal degradation, possibly through CKI and GSK-3β, which are involved in β-catenin phosphorylation on key amino-terminal serine residues [236], since β-catenin is free to exert its functions in the absence of a functional p53 pathway
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