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This precise, site‐specific modulation of EGFR signaling by CAP suggests it may function as a paralogous modulator to estrogen, capable of fine‐tuning complex receptor tyrosine kinase–hormone receptor interactions that are implicated in both pain perception and tissue pathology
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First, most studies have focused on the tumor-specific effects, and the long-term impact of these modulators on systemic redox balance remains unknown
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